鼠三叉神经节中神经肽阳性胞体的分布与密度

作者:刘向辉 斯方杰 徐正林 饶志仁  文章来源:临床口腔医学杂志 

2008-7-7 15:49:57         【博客】 【论坛】 【投稿】 【打印】 【关闭


  本实验对TrG中SP、CGRP、NPY阳性反应的观察将有助于进一步研究神经肽在口腔颌面部病理状态下的作用机制。■

作者单位:刘向辉(解放军81医院 南京,210002)
     徐正林(解放军81医院 南京,210002)
     斯方杰(第四军医大学口腔医学院)
     饶志仁(神经生物研究所)

参考文献:

[1]艾民康.神经生物学基础.第一版,同济医科大学出版社,1992;220~234,259~266.
[2]Hargreaves KM,Swift JQ,Roszkowsk MT et al.Pharmacology of peripheral neuropeptide and inflammatory mediator release.Oral Surg Oral Med Oral Pathol.1994;78:503.
[3]Sigvard K.The influence of neuropeptides,serotonin and interleukin l β on temporomandibular joint pain and inflammation.J Oral maxillofac Surg,1998;56:189~191.
[4]Maarten P.Frans W,Baljet B et al.Calcitonin gene related peptide and substance P immunoreactivity in the monkey trigeminal ganglion,an electron microscopic study.Brain Res,1993;629;315~318.
[5]Nikolail T,Thame K.Distribution of calcitonin gene related peptide-and neuropeptide Y-like immunoreactivity in the trigeminal ganglion and mesencephalic trigeminal nucleus of the cat.Acta Histochem.1995;97:213~223
[6]Salaki Y.Effects of peripheral axotomy of the inferior alveolar nerve on the level of neuropeptide Y in rat trigeminal primary afferent neurons.Brain Res,1994;664:108~114.
[7]Holzer P.Local effector functions of capsaicin sensitive sensory nerve endings:tachykinins,calcitonin gene related peptide and other neuropeptides.Neuroscience.1988;24:739~769.
[8]Lembeck F,Holzer P.Substance P as a neurogenic mediator of antidromic vasodilation and neurogenic plasma extravasation.Arch Pharmacol,1979;310:175~183.
[9]Lundberg JM,Lars T,Tomas H et al.Neuropeptide Y(NPY)-like immunoreactivity in peripheral noradrenergic neurons and effects of NPY on sympathetic function.Acta Physiol Scand.1982;116:477~480.
[10]Lofz M、Vaughan J、Carson D.Effect of neuropeptides on production of inflammatory cytokines by human monocytes.Science.1998;241:1218~1221.
[11]Edward S,Francis J,Jeffry L.Substance P.neurokinin A and neurokinin B induce generation of lL-1-like activity in P388 D cells,possible relevance to arthritic disease.J lmmuol.1988;141:3546~3569.
[12]Lofz M,Carson D,Vaughan J.Substance P activation of rheumatoid synoviocytes:neural pathway in pathogenesis of arthritis.Science.1987;235:893~895.

本新闻共3页,当前在第3页  1  2  3  

责任编辑:姚红祥  

上篇文章:用山羊建立下颌牵张成骨实验动物模型
下篇文章:分子外科缝线术的建立及其优化方案的研究